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AN EXTRACT FROM: CHEMICAL SENSITIVITY VOLUME 1 BY PROFESSOR WILLIAM J. REA. PUBLISHED 1992. * Human Toxicity Excerpts RE: butoxyethanol * Full article
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Resources
for the Chemically Injured - Top
Free Images Free MIDI Hymns LassenTechnologies' Top LinksLaboratories: These are laboratories which can help the chemically sensitive and his/her doctor.
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http://www.snowcrest.net/lassen/mcsei.html ________________________________________________________________________ NASA - Research May Help - Those whose DNA has been chemically altered - In Danger of Cancers |
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"We need to pay a lot more attention to preventive medicine-
-not so much to radiating, poisoning and cutting." -Dr. Ben Carson *
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Information on 2-butoxyethanol, via the web:
Here are the urls:
International Chemical safety card: http://www.cdc.gov/niosh/ipcs/ipcs0059.html
a brief summation here:
| 2-BUTOXYETHANOL | ICSC: 0059 |
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Ethylene glycol monobutyl ether Monobutyl glycol ether C6H14O2/CH3(CH2)2CH2OCH2CH2OH Molecular mass: 118.2 RTECS # KJ8575000 ICSC # 0059 UN # 2369 EC # 603-014-00-0 |
| ENVIRONMENTAL DATA |
This substance may be hazardous to the environment; special attention should be given to the water environment and aquifer. | |
Note: What is the most minimum exposure that could cause such?
EFFECTS OF SHORT-TERM EXPOSURE:
The substance irritates
the eyes, the skin, and the respiratory tract.
Exposure could cause central nervous system depression and
liver and kidney damage.
NOTE: with daily urinalyses testing, how could exposures be allowed
to reach this point?
EFFECTS OF LONG-TERM OR REPEATED EXPOSURE:
The liquid defats the skin.
The substance may have effects on the haematopoietic system , resulting in
blood disorders.
such
as *hemolytic
anemia; and kidney damage (Agency for Toxic Substances and Disease
Registry 1998). * hemolytic
anemia in this case would be premature destruction of red blood cells caused
by chemical poisoning
Also check http://www.who.int/pcs/cicad.summaries/cicad_10.htm
and a 50-page document from the WHO:
http://www.inchem.org/documents/cicads/cicads/cicad10.htm
Studies on Central Nervous System affected by solvent exposures: http://www.whis.nzl.org/snftaas/pt17.html
It is important for those affected to have a voice and know that they do not stand alone.
- GASS (Glut, Aldehyde, Solvent Support a support network for the aldehyde and solvent affected) was established in 1997 initially to help people affected by glutaraldehyde and the wider cocktail of harmful X-ray processing chemicals - including sulphur dioxide, acetic acid, potassium hydroxide, hydroquinone, diethylene glycol, butyraldehyde, toluene/benzene, formaldehyde or other aldehydes.
- Professor Bill Glass. Christchurch. Article on Solvent Neurotoxicity
- Article on Solvent Neurotoxicity by Safeguard Magazine
- Article on Organic Solvent Neurotoxicity by J. Donald Millar, M.D., D.T.P.H. (Lond.) Assistant Surgeon General Director, National Institute for Occupational Safety and Health Centers for Disease Control " ... Studies have demonstrated that these effects can persist for months to years after removal of workers from solvent exposure. The extent to which chronic neurotoxicity is reversible remains to be established; peripheral nerves have the capacity to regenerate, but damage to the Central Nervous System is more often permanent."
- Chemical Exposure and Disease - Lots of Links to interesting and informative articles about Solvent Neurotoxicity.
- OSH-world - for all fields related to work injury, safety, and health worldwide.
- CDANET A Discussion/Chat/Mutual help list run by and for the CHEMICALLY DISABLED.
- An Island Place
- Canadian Neurotoxicity Information Network
| Although
SNFTAAS now GASS initially focussed on the health industry's use
of glutaraldehyde (also a sterilant e.g. Cidex, for medical
instruments), it became increasingly obvious that a
chemical support network was needed.
It has happened and GASS now exits. Many of the effects of toxic chemicals are similar and frequently those affected are accused of malingering; of it being "all in the mind", pre-, mid- or post-menopausal, stress related, or from depression or an "illness belief". Those affected are often misunderstood or ridiculed or subjected to enormous pressure to return to work before they are able (if at all). Symptoms (most common first) include: headache, sore throat, voice change or loss, constant humph, catarrh, unexpected tiredness, fatigue, sore/watery/gritty eyes, irritability, anxiety, depression, loss of concentration, short-term memory loss, reactions to other chemicals, dizziness, blocked nose, rhinitis, mouth ulcers, bad/metallic taste in mouth, lip sores, chest tightness, shortness of breath, chest pains, asthma, sinusitis, nasal discharge, nose bleeds, ulceration of nasal mucosa, itchy burning skin, rashes, dermatitis, aching joints and muscles, fibromyalgia, uncontrollable twitching, heart arrhythmia's, palpitations, abdominal pain, nausea, diarrhea, tinnitus, earache, pins and needles, numbness in fingers and toes, Raynaud’s Syndrome, menstrual irregularities, pain/difficulty urinating. Other areas of concern include liver damage, cancer, teeth and gum problems, reproductive effects and lack of interest in sex. |
"A low intake of antioxidants was associated with an increased risk of all gastric cancer subtypes and a high parallel intake of ascorbic acid, ß-carotene and [alpha]-tocopherol was significantly associated with reduced risk, suggesting synergistic mechanisms. Our findings also imply that other bioactive compounds in fruit and vegetables could be important in reducing the risk of gastric cancer. Exposure to metal work and phenoxy acids was associated with increased risk of gastric adenocarcinoma, but causality is unclear. With a more correct classification of exposure, the risk associated with H. pylori appears stronger than earlier, confined to non-cardia tumors, and substantial for both intestinal and diffuse type tumors. Interactions between antioxidants, H. pylori infection and cigarette smoking may considerably modify the risk of all types of gastric adenocarcinoma, indicating that H. pylori induces oxidative stress that could be ameliorated by dietary antioxidants." http://diss.kib.ki.se/2000/91-628-4066-5/
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11-14-03 restate