Corrections for web pages on comments at Gulfwarvets.com

Corrected posts - January, 2004

Feb, 2004  -  March, 2004

My trip to Neuro at VA Houston Hospital - enough is enough

MM Reply posted March 05, 2004


It's disgusting. Doctors should care more, and if they are not interested, bow out in the first place, let another doctor who is interested, or more knowledgeable see you.

How much will they learn by only checking the blood for cholesterol? The blood has a lot more to tell, like why the fatigue & if something more may be starting up. (Absolute cell count; retic ratio - how hard is it to check? and liver and kidney function, too?)

In our town, we have doctors that must work for the big industry or someone besides the victims of oil/chemical exposure. I spoke to one of the doctors a year ago, expressing concern over a young man harmed by the chemicals of the Exxon Valdez oil spill cleanup, to which the doctor said point blank, "No one was harmed by the oil or the chemicals, either" Sad story. Guess you weren't hurt in your military service, either, right!

This doctor also said a man who died in 1998 died of leukemia. At time of death his RBCs were 2.99 (Normal range is 4.5-6); WBCs were 1 (normal range is 4.5-10.7); PLT were 22 (Normal range here is 150-450)

Maybe another doctor should give an opinion of the medical file, and change the death certificate to death by aplastic anemia.

This man only had one week's exposure to fresh oil in March, 1989. That's when the benzene is most volatile and most health damaging.

In his case, he was said to have died from leukemia. But wouldn't there be elevated white blood cell counts, if so?

Others die of kidney failure, but the death certificate will say stroke or heart attack.

4-3-04 To get to the main point: The chemical that harmed the EVOS workers is one that gulf war vets were exposed to. Note the first link on this page

Now, until you find out what the retic ratio is, and the size and shape of the red blood cells, you don't know whether or not there is harm to your red blood cells that underlies all the neuro and other symptoms you name. Blood counts and other stuff won't tell you. So, don't stop short of checking this out.

posted April 06, 2004:

I was wondering whether there is a difference in treatment when diabetes is actually a reflection of endocrine disruption & not merely the regular diabetes people get as they get older (insulin resistant, as you say)

I've been noticing more carefully, as well, the definitions of CFS & CFID and it looks to me like the docs don't know the cause of this fatigue & just give it this label.

Doesn't it look like they are describing 'gulf war syndrome' symptoms? That's what it looks like to me.

posted April 07, 2004:
Thank you for sharing, dd

My theory is that the immune system is attacking the red blood cells (of course, sometimes there is infection to deal with); but that the blood is actually damaged - and that's the main cause of lack of oxygen.

Other organs under siege? The thyroid being out of whack, I've heard several times from Gulf War vets. It fits the 'pattern' of endocrine disruption & also in itself would cause an extreme exhaustion, I've been told. Any of these acute effects? Or these organ/systems? Or several of these?

Some of the research can be accessed by medical doctors at no cost to them. Some research summaries ACTUAL ENTRY IN RTECS FOR COMPLETE INFORMATION.

Isn't weight and growth also glandular functions? ... in addition to just diet and exercise? Such as possibly the thyroid and the hypothalamus? I saw a comment from a 'lean, mean Marine' gulf war vet somewhere in which he had gained an immense amount of weight and the doctor walked out with no help of any kind for him. Maybe, dd, you are the 'catalyst' for finding more answers than most people can - because you have doctors who are interested and who are working with you! Maybe you are 'fighting the war' for health justice for all of the military?

Have you had this TRH test?

Glad to hear your prognosis is good; I should be doing the same - going to the gym & loosing weight. Right now walking half a mile to town and back again each day is an add on to my 'exercise'

I've heard that there is something in the heels of one's feet that benefits by walking, etc & helps pump blood in your system.

Immunoglobulin A, B, and M what are they?

"Just a quick question for all the guru's out there. My value for M was really low and highlighted in bold by the lab. The range value is 40 to 230 and mine was 29 L. Just wondering what all that means." from a gulf war vet
posted March 05, 2004:
From a definition found on the internet:
quote:
Immunoglobulin A Deficiency:
Synonyms and related keywords: immunodeficiency, selective IgA deficiency, selective immunoglobulin A deficiency, primary antibody deficiencies, primary antibody deficiency, antibody deficiency, IgAD, immunodeficiency disease, sinopulmonary infection, sinus infection, otitis media, stomach cancer, Giardia lamblia, G lamblia, Helicobacter pylori, H pylori, GI cancer, gastrointestinal disease, GI disease, Crohn disease, upper respiratory tract infection, lower respiratory tract infection, chronic diarrhea, transfusion complication, blood product reaction, adverse transfusion reaction
To continue: "Secretory IgA antibodies can neutralize viruses, bind toxins, agglutinate bacteria, prevent bacteria from binding to mucosal epithelial cells, and bind to various food antigens, thus preventing their entry into the general circulation. The role of serum IgA is unclear."

"IgAD is a primary immunodeficiency disease presumed to result from a failure of terminal differentiation in IgA-positive B cells. Multipotent hematopoietic stem cells give rise to progenitors of T cells, B cells, and natural killer cells."

"The development of B-lineage cells begins in the fetal liver. B-lineage cell development then transfers to the bone marrow when it becomes the major hematopoietic organ. Pre–B cells become immature immunoglobulin M (IgM)–positive B cells and then migrate from the bone marrow to lymph node germinal centers. After leaving the bone marrow, the B cells mature and express immunoglobulin D receptors, respond to antigens, and, with the help of T cells (CD4+), undergo proliferation and plasma cell differentiation (International Union of Immunological Societies, 1999)."

Source: http://www.emedicine.com/med/topic1159.htm

posted March 05, 2004
:
In sharing your question with 'Mike' his comments are:

"The immunoglobulin molecule: That's a rather complex molecule! Well, the lymph nodes crank out this protein molecule to go after foreign bodies in an organism.

Too low numbers found in a blood test just means that particular means of defense is too low, possibly in decline.

Something is impeding the lymphs; bad nutrition, poisoning, old age, etc. Trick is to discover what caused such a condition.
I suspect you're on the path to connect that ester-alcohol solvent C6H14O2/CH3(CH2)2CH2OCH2CH2OH to lymph damage. Since the liver is definitely damaged with that substance just like longterm metalysis of ethanol, follows other organs will suffer as well."

I met Mike Simmons when he gave permission to use some of his 'pianodude' music to the website I was working on.

Then he added this valdezlink.com/media-ak/Exxon_Valdez_Movie.swf

Turns out he knows about various chemicals and their effects, too!

"Then someone explain to me Igg. Had high Igg to epstien barr 960, and high Igg to herpes 1 or 2 600+. I was told these are intibodies to the viruses. And that Igm would indicate current active infection." Pam, a gulf war vet
posted March 07, 2004:
Pam, is this the same thing?
quote:
IgG: Immunoglobulin G, a major class of immunoglobulins found in the blood, including many of the most common antibodies circulating in the blood. Also known as gamma globulin.
MERCK medical dictionary has some pretty good definitions, check this chapter on immune: THE MERCK MANUAL, Sec. 12, Ch. 146, Biology Of The Immune System and

THE MERCK MANUAL, Sec. 11, Ch. 145, Aids-Associated Hematologic Disorders And Malignancies

If the level is high, then your immune system is doing pretty well? You can help everyone out by checking into it and letting us know if you learn something more

posted March 07, 2004:
PS

Could this be an autoimmune thing going on?

If your system is out of balance, such as what I expect to be found eventually for those with hemolytic anemia from 2-butoxyethanol or diethylene glycol monobutyl ether ...

I would expect it to be diagnosed as acquired autoimmune hemolytic anemia

How I wish I'd realized then ... that this is PRIMARILY an AUTOIMMUNE problem

... first with the red blood cells ... and then AUTOIMMUNE lots of other things

Check these extra things to find it out

Many 'syndromes' fit the pattern of what EGBE does

What does it look like

Cold agglutinin disease is an acquired autoimmune hemolytic anemia due to an IgM autoantibody usually directed against the I antigen on red blood cells.

AIHA

Autoimmune hemolytic anemia. Occurs when the body produces antibodies that coat red blood cells.

Immune System Activated Coagulopathy:  Thickening of the blood caused by Immune System

etc  etc  etc

What about hemoglobinuria?  

July 5, 2005

e-mail

Small Red Blood cells?

Does EPA think EGBE is no big deal? *

Must be good lobbying of chemical companies -

Not good nor is 2-(2-butoxyethanol)    DGME

ETC